The Arlene Berry Death Coverup: Update 2009 (Part 2)
Notably, the record at A-24 documents a HR (heart rate) of 174 bpm at 0320 hours that is consistent with an "awake intubation", (any suspicion of difficulty intubating, for any reason), marked by panic with "awareness".
To illustrate, the Vital Signs Record at A-24 documents a heart rate of 174 bpm at 0330 that is consistent with "trauma", while the Ventilation Record seen at A-16 documents a heart rate of only 126 at the very same time, a significant difference, suggesting that the timeline for that event was in fact altered by the Ventilatory Therapist to obfuscate iatrogenic trauma related injury. The Vital Signs Record is a mechanical record with a run time, while the Ventilation Record is a handwritten account, mared by having been rewritten. Which is more likely to make copious errors or downplay an event by omission or telling lies?
There is nothing on record to suggest that anesthesia was or wasn't given to prepare the patient for the intubation procedure. My opinion is that if its not on record it didn't happen.
The earliest indication of shock is an increase in heart rate (HR).
According to Dr. Jordan "the intubation proceeded uneventfully", while N-2 of the record documents the ET (endotrachial tube) "pulled back 4 cm" at 0425 hours. From that record it seems clear that the endotrachial tube had been malpositioned for almost one full hour before the error was discovered by one of the nurses, as evidenced by the record at N-2; infers negligence on the part of the Dr. Jordan, including failure on his part to identify an incorrectly placed airway in a timely manner. Both myself and the patient's foster brother were present to witness that event.
Malpositioning of ET tube can cause airway obstructionand may also result in tissue trauma, andbleeding. When an endotrachial tube is misplaced in the esophagus and misplacement is detected late, a compromiseof the patient's safety can be significant.
A-12 of the medical record documents a blood pressure of 163/117 bpm at 03:20 hours that by 03:45 hours had dropped to 85/58, following intubation, with an additional drop to 85/52 bpm by 3:52 hours, over a span of some 7 minutes, as evidenced at N-2 in the Nurses' Notes.
A-17 documents "being suctioned for moderate amounts of coffee-ground emesis by RN" at 0330 hours. Gastrointestinal bleeding due to stress ulceration is also an important complication in critically ill patients. GI bleeding is a medical emergency that was basically ignored by the healthcare providers, in this case.
Stupor and coma are characterized by impairment of the arousal system. In stupor, a person arouses only in response to strong verbalor tactile stimuli, awakens briefly, and then lapses back into a sleeplike state after the stimulation stops. In coma, a person cannot be roused to consciousness. With GBS the patient is conscious but unable to respond due to a severely paralysed motor function.
Besides dangerous cardiac manifestations, neuroendocrine changes are also reported and could induce electrolytes and fluid balance impairments. Polyuria has been observed in a severe case of GBS. Polyuria in GBS is multifactorial and would be partly due to a dysregulation of osmoreceptors.
The physician's Lab Work Summary at A-19 documents the charting of a course of Hematology (bloodwork) and Coagulation. The same record documents a Fibrinogen level of 4.67 H (the normal range is 2.00-4.00). Elevated fibrinogen levels induce a state of hypercoagulability.
When blood protein is high, CSF usually clots because of the presence of increased fibrinogen. Serum fibrinogen levels in a safe range is <300 mg/dL. The plasma fibrinogen level appears to reflect disease activity in acute Guillain-Barré syndrome and is typically elevated at presentation. In fact, ongoing activity of Guillain-Barré syndrome may be reflected by a persistently elevated fibrinogen level. Acute phase reactant: marker of inflammation Further, elevated levels may also be seen with TRAUMA of any kind.
A-19 documents a D-dimer test level of 1000 H (<500)(fibrinogen and d-dimer correlates with thrombotic activity) suggests thrombosis. Thrombosis signifies the formation of blood clotting within vessels of the brain or neck. People who are suffering from a severe infection are more likely to develop dangerous blood clots, but inappropriate combinations of medications or treatment can sometimes be the worst offenders.
The Cardiac Index at A-18 documents the patient's ventillation rate at 129 bpm (breaths per minute) at 0417 hours, with heart and breath rate increased. Increased heart and breath rate can suggest clinical insult, such as caused or worsened by medications, resulting in oxygen deprivation. The Cardiac Index documents the patient’s age at "55 years", she was only 41 at the time of her death; can imply negligence, or even patient record swapping.
A-19 documents the aPTT= activated Partial Thromboplastin Time, a test used to determine the efficacy of various clotting factors used in the diagnosis of coagulation disorders documents the therapeutic range for heparin therapy at 60-100 seconds (23-35 is the normal). The time of that assessment was documented at 0400 hours. The aPTT is typically elevated in 90% of those with coagulopathy.
A-19 documents a "PLT ESTIMATE" - "MOD INCREASE" confirming a moderate increase in platelet aggregation activity (blood platelets sticking together), indicating that blood thinners may be needed to prevent blood clots.
The ambulance call report seen at N-7, of the Nurses' Notes documents that the patient was intubated and vented and that she was seen to be "stable", but that she appeared to be "pale, dry and cool". In patients with GBS, the skin may become pale and dry, and sweating may become reduced. Body temperature can also be marred by the effects of drug-induced temperature dysregulation, which can suppress sweating, causing central nervous system impairment, often resulting in an afebrile state. Cool, dry skin can also suggest late sepsis, a prominant finding with iatrogenic neglect.
Hemodynamic instability has also been defined more broadly as global or regional perfusion that is not adequate to support normal organ function.This definition recognizes the obligation to insure adequate organ perfusion during the flaccid period in patients with GBS.
According to the Nurses' Notes at N-1 of the record the patient was given Gravol 50 mg x 10by paramedics at 0620 hours, while the record at N-7with respect to medications documents "See Nsg Notes". Gravol (dimenhydrinate) is contraindicated in lung disease and has also been reported to "mask the presence of underlying organic abnormalities and/or the toxic effects of other drugs".
The record at A-8 and A-9 documents "Medi-Vac team were due to arrive at 0435", while the Ambulance Call Sheet documents "call received at 0620" hours, a significant difference. The record at N-7, documents "pulses X 4 good"; head and neck OK; chest OK; "abdomen OK"; pelvis OK; extremities OK.
Following her transfer to Sudbury on May 24th of 2000, Arlene Berry's remains was returned to Kirkland Lake several days after family had been notified of her death via her foster brother acting as a family contact, who was notified by phone. On seeing the deceased vicim, her eyes were "sunken in appearance", with swelling and distortion of the face, eyes, and lips, as was the case, marked by a rash-like redness resembling a sunburn with "blistering" wrinkles in the skin in the area just below the right eye, consistent with a delayed hypersensitivity reaction, or fixed drug-eruption, evidenced by all who attended Arlene Berry's viewing at the Monette Funeral Chapel in Kirkland Lake.
It is believed that Arlene Berry's death was deliberately provoked and that her eyes were taken by Drs. Sauvé, and Adegbite at the Sudbury Regional Hospital, upon remote third party consent (foster brother), utilizing deception to obtain that consent, by-passing permission from Arlene Berry's immediate family, ie, her de facto common-law spouse and her children. Only biased clinicians might provide less aggressive scrutiny, withhold medical treatment and influence the family in inappropriate ways such as this. Indeed, the fraudulent taking of the patient's eyes in the manner in which it was done can only be construed as theft. From the information at hand, it seems clear that Drs. Sauve and Adegbite sought to open the way, under misleading conditions (influence of drugs, and metabolic disturbances) to organ donation from brain death. The diagnosis of brain death allows organ donation or withdrawal of life support. These doctors allowed this patient to die to achieve their nefarious ends. Certifying brain death to cover-up medical blunders or to increase organ donations constitutes murder.
At a first meeting with the coroner held at the OPP Detachment in Kirkland Lake sometime in July of 2001, Dr. Barry A. McLellan, who was the Regional Supervising Coroner for northeastern Ontario at the time admitted to family that there was "no evidence on record to suggest matastasis", meaning spread of cancer. In fact, there is nothing at all on this record to support a diagnosis of cancer in the first place. For the record, fungal, mycobacterial, parasitic, and indolent bacterial infections heve been known to occasionally mimic cancer. In fact, certain pulmonary infections can mimic pulmonary neoplasms.
At a subsequent meeting between family and Dr. McLellan, he provided us with a view of a CT scan that was purportedly done in Sudbury, Ontario at the time Arlene's death on May 24thy of 2000, although I suspect it may have been done following withdrawal of llife support. It shows multiple lesions of undetermined origin. The pathogenesis of these yet undetermined lesions remaines unclear but a metabolic disorder seems the most plausible pathological factor. Based on the patient's belated CBC's, the lesions are consistent with collections of purulent exudates (pus producing bacteria), suggested by an elevated Neutrophil count, and/or pockets of pooled blood, as suggested by an elevated Fibrinogen in the presence of an elevated D-dimer, a hallmark of thrombus formation, ie., blood clots. The enhancement is obviously due to infection, or blood pooling, or both.
A-1 of the record also documents "she died several days later with numerous metastatic lesions to her brain". According to her death certificate, Arlene Berry died May 24th of 2000, the very same day she was transferred out to Sudbury. As to the cause of death, according to a Dr. Sauve in Sudbury, she died "meeting brain death criteria".
No attempt to diagnose was made at that time, or at all. No pathalogical reason was given for the declaration of brain death. No process of exclusion was undertaken without which a diagnosis of brain-death should never have been considered.
No autopsy was performed. No appropriate period of observation and/or trial of therapy was ever undertaken. In fact, Arlene Berry was rushed to her death within five and one-half hours of her departure from Kirkland Lake to Sudbury, some 210 miles away. One might ask how much time did these medical dolts actually spend assessing the patient before pulling the plug on her?
Although many conditions can mimic brain death clinically upon examination, without excluding them you will KILL a person by homicide, or criminal negligence, despite the reversibility of brain damage.
Brain death is defined as the irreversible cessation of function of the entire brain with three specific criteria: 1) coma, 2) absent brainstem reflexes and 3) apnea. In addition to these clinical criteria, there are important prerequisites: 1) NO intoxication or poisoning, 2) NO core temperature greater than 32 degrees Celsius,3) clinical or neuroimaging evidence of acute central nervous system catastrophe and 4) absence of confounding medical conditions such as severe electrolyte, acid-base, or endocrine disturbances.
As a safeguard in determining brain death a number of tests need to be carried out every 6 hours and recorded, the physicians performing this determination must not be part of a transplantation team. In some cases, 48 to 72 hours is required to evaluate brain death and a repeat examination with observation up to an additional 24 hours is sometime needed. The length of time between serial examinations to declare brain death varies marginally from 6 to 72 hours.
Notably the Sudbury doctors involved were a part of an organ harvesting and transplantation team. It was Dr. Sauve who, utilizing deception, sought to obtain permission from a remote party to obtain the victim's eyes, without immediate family knowledge or consent.
With respect to the initial CT scanhereinbefore mentioned, according to the coroner's expert "in the right occipital region there is a spot that measures less than 1 cm that is consistent in appearance with either a small hemorrhage or perhaps a small metastatic tumor". He could only speculate. NO biopsy was done (a biopsy, is required for obtaining tissue for pathological confirmation of the diagnosis). The solitary lesion is also consistent in appearance with an abscess secondary to an occipital dermoid cyst, or early stage cerebritis during/after capsule formation in the early stage of abscess development. Rupture of a dermoid and leakage of a cyst contents into a ventricle or subarachnoid space may produce an epidymitis or meningitis respectively. Further, capsules can rupture resulting in the formation of multiple abscesses.
The bald truth is that localizing signs of brain tumor include a loss of visionon the side of an occipital neoplasm. Compare occipital abscess, or pyogenic brain abscess, usually of bacterial origin. The occipital lobes interpret vision. Brain tumors are more solid/dense and therefore are usually associated with multi-focal deficits; tumors of the occipital lobe usually produce homonymous hemianopia or partial visual field deficits. Had the lesion been a recent tumor, there would have been onset visual misperception in half of one or both visual fields, with visual impairment and subsequent loss of vision with evolution, prior to hospital admission. That did not happen.
Know that even multiple brain abscesses may not cause focal deficit to suggest their presence. Non-neoplastic demyelinating processes of the brain with ring enhancing lesions and mass effect on MRI imaging, mimicking malignant brain tumors, are rare phenomena. The radiologic appearance of demyelinating pseudotumors as contrast-enhancing solitary masses that mimic tumor is well documented, ie., "tumor-like" masses of demyelination, or granulocytes that mimic brain tumors (granulocytes, which are cancer killing cells turn black as they die), or infection. Further, with multiple abscesses or infection the meninges typically show a purulent exudate that obscures the sulci making radiographic appearance of microabscesses less visible, hence they are "not well opacified". Morbidity due to a brain abscess generally results from brain herniation due to mass effect, the result of iatrogenic neglect, or substandard care.
No autopsy was performed. Further, a family request for a formal inquest was also denied. Dr. McLellan concluded that Arlene Berry had died of natural causes suggestive of metastatic CA of the brain with multiple brain tumors after eliciting what can only be construed as a most questionable opinion from one of his fellow colleagues believed to be associated with the Sunnybrook Health Sciences Centre, where McLellan spearheaded the North Telehealth Network. However, the medical record of Arlene Berry for May 23rd and 24th of 2000, tells a very different story from the opinion postulated.
Eleven percent of mass lesions in cancer patients are not metastases; mass lesions that can masquerade as brain metastasis include abscess(20%) and granuloma (less common and mostly associated with mycobacterial or fungal infection). The commonly observed deficits observed in CNS infection include weakness on one side of the body (hemiparesis), impaired speech production (dysphasia), visual field deficits (may or may not be present), and an inability to smoothly coordinate muscle movements, such as during walking (ataxia).
Patients with a dioagnosis of primary or metastic brain tumor(s) associated with a CNS event should have a meticulous review of their history for other possible causes. Brain Tumor is often considered in the diagnosis of lesions demonstrated on brain computed tomography (CT) and/or magnetic resonance imaging (MRI). Brain tumors usually show abnormal densities on CT or altered signal intensities on MRI, mass effect, and sometimes contrast enhancement after the intravenous administration of contrast material. Lesions with these features, however, are not always brain tumors and establishing the diagnosis of a brain tumour is not always a straightforward process. Cerebrovascular diseases, demyelinating processes, inflammatory or infectious diseases, and other miscellaneous diseases can show similar imaging findings.
Many non-neoplastic neurological diseases can mimic brain neoplasms on neuroimaging or on histological examination, including multiple sclerosis, stroke, pyogenic abscess, toxoplasmosis, tuberculosis, cysticercosis, fungal infections, syphilis, sarcoidosis, Behçet disease, radiation necrosis, venous thrombosis, Guillain Barre syndrome and others. Multiple intracerebral hematomas can also mimic brain metastases. West Nile virus encephalitis mimicking central nervous system metastases from small cell lung cancer is reported in the literature.
The investigation and analysis has been ongoing for almost 9 years with everything pointing to possible GBS, with two second-guess possibilities in the differential which can not be ruled out, namely West Nile Virus, or Botulism, in that order. The differential diagnosis can be difficult, because the symptoms mimic those of Guillain-Barre syndrome. Further, fever in WNV apparently does not present in all cases. With GBS, fever is usually absent at onset. Although brain death mimickery is not an acute finding in WNV, it is with the Miller Fisher variant of GBS, a brainstem encephalitis which also mimics brain death. That botulism may also mimic brain death needs always to be kept in mind. Yet, meningitis, encephalitis and Guillain-Barre syndrome remains undeniably the presentation in this case. I have to ask, is there a common denominator here?
Meningitis and encephalitis are usually caused by viruses or bacteria. Less commonly, encephalitis can result from a bacterial infection, such as bacterial meningitis, or it may be a complication of other infectious diseases. Guillain-Barré is preceded by a viral or bacterial infection. Viral meningitis is the most common cause of aseptic meningitis, an inflammatory process involving the meninges in which usual bacterial etiologies cannot be identified.
The opinionated author (whose signature was erased) perceives himself to be expert in his field. Believed to be a neurosurgeon, he is generally lacking. The bald truth is that specialists in a given field are not always expert in that field. For the record, his opinion remains unsubstantiated. He also failed to provide any evidence whatsoever which might support a finding of metastatic CA of the brain. Further, a paltry CT scan does NOT provide conclusive proof of metastatic brain tumors. For that it takes a biopsy and in this case that was never done. Therefore the true nature of the lesions were never established. Nor does a CT provide conclusive proof of brain death. In fact the author who rendered the opinion suggesting metastatic cancer is pretty much off-the-wall; what one might expect from a "hand-picked" self-interest cash for comment shill, or someone to one equated with a first year medical student who is thus unworthy of belief.
Further, the opinions expressed by the nameless author admits that his opinion "does not take into account all the facts and circumstances surrounding the patient's death", being his disclaimer. Such a disclaimer also raises doubt about the accuracy of the opinion. He bases his opinion on assumption, while ignoring the facts. Obviously he did not take the time to study the patient's medical record. Instead he opted to tailor or lard his report to justify the opinion that was, in his opinion , being sought by Dr. McLellan for the sake of expediency. This infers a blatant attempt by all concerned to obfuscate the truth. A finding of possible meningitis with abscess of the brain and/or multiple intracerebral hematomas would have given more credibility to his opinion. In fact, some of the opinion expressed by the nameless author could also be used to support a finding of meningitis. There is absolutely nothing on record to support a finding of metastatic cancer of the brain, apart from a paltry CT, with no evidence on record to support it.
Rapid deterioration is an invariable accompaniment of an untreated condition, in this case, an undiagnosed case of fulminant GBS with overlaping meningitis, in which rapid progress of the disease may actually be displaying a pronounced"blood-brain barrier breach", characterized clinically by "rapid evolution", the result of totally inappropriate treatment, and medications which can breach blood-brain barrier integrity, ie. the drug Stemetil. Breakdown of the blood-brain barrier usually precedes inflammatory demyelination. GBS is often associated with cerebrospinal meningitis and encephalitis. Coma in GBS is rare and is the symptom of cerebrospinal meningitis and encephalitis. The clinical manifestations of this condition included areflexia in the cranial and spinal nerves as well as apnoea. Cisterns may contain pus in cases of meningitis or other inflammatory conditions, such as sarcoid, or demyelination.
Any medical professional who, in rendering his opinion as to a cause of death, volunteers his unsolicited opinion as to another doctor’s conduct or standard of care in the face of overwhelming evidence to the contrary, finding in favour of his fellow colleague(s), invites being labeled retrospectively with the most nefarious motives.
Although two physicians with experience and expertise must be responsible for the declaration of brain death, and a neurologic condition capable of causing brain death is a mandatory prerequisite to the diagnosis of brain death, there are reports in the literature of conditions that mimic brain death or that provide examples of the mistaken diagnosis of brain death.
The clinical diagnosis of brain death occurred after the patient had received an IV tranquilizing agent and while still under the influence of morphine sulfate, an opiate narcotic.
GBS is one of the few neurological diseases whose clinical manifestations may mimic or appear to be identical to those in brainstem death, illustrating an extreme polyneuropathy.
Canadian standards do not test function of the "entire brain", and there is sound evidence that many individuals who meet the clinical criteria of brain death continue to have some cortical, subcortical, or brain stem function. Many patients diagnosed as brain dead do not have hemodynamic collapse, they have physical findings such as bowel sounds, and are reported to have autonomic reflexes (tachycardia and hypertension) at the time of organ retrieval, suggesting a horrific death.
The clinical diagnosis of brain death in this case was made in the presence of metabolic derangements and endocrine abnormalities and constitutes an act of wanton and reckless disregard for human life. There have been many challenges to the several concepts of "brain death" and the means of their diagnosis worldwide (vide infra). Indeed, it seems that there is now an emerging consensus that "brain death" diagnosed by any of the protocols in current use worldwide is "not death."
After the diagnosis of brain death, the focus of patient care shifts from interventions aimed at saving the patient's life to interventions aimed at maintaining viability of potentially transplantable organs. Given that current clinical testing does not assess subcortical brain function, ‘whole brain death’ cannot be conclusively identified at the bedside by using clinical criteria, and most certainly not on the basis of a paltry CT scan. The CT is useful only in pretty severe cases, such as head trauma and/or during the few days after an anoxic (lack of oxygen) brain injury and also in cases where there has been a massive stroke. If the question is ischemic injury [brain damage caused by lack of blood/oxygen to part of the brain] you want an MRI and PET. For subsequent evaluation of brain injury, especially brain death, the CT is pretty much useless.
Many Ontario physicians actively involved in the identification of brain death, if the truth be known, are unable to identify the requisite diagnostic components of brain death, and/or are unable to apply the criteria correctly. Some physicians will take advantage of the situation to perpetrate a medical murder in order to harvest organs from potential donors, while others will simply declare "brain death" in order to avoid liability for medical wrongdoing.
Organ transplantation is premised on professional and public acceptance that the donor is dead and that the cessation of all brain function persists for an appropriate period of observation. The diagnosis of brain death allows organ donation or withdrawal of life support. The diagnosis of brain death is dependent upon the exclusion of certain medical conditions without which the diagnosis of brain-death cannot be considered and influence of medications is one of them.
Brain death declaration may be described as an esoteric creation of neurologists and neurosurgeons who are seeking to speed up death for the purposes of an organ transplants. In this case, the harried and hurried physicians did not rely on those esoteric criteria in pronouncing the patient "dead." They simply used the questionable "brain death criteria" (based on a paltry CT after withdrawal of ventillatory support) as a pretext to operate and remove their patient's eyes while she was still very much alive.
There are numerous reports of brain death declaration and at least two where the criteria were deliberately misrepresented, or obfuscated in an attempt to retrieve an organ for transplantation, as in this case.
Brain death: resolving inconsistencies in the ethical declaration of ...
Purpose: The first criteria for the determination of brain death were developed ... two cases were presented where the criteria were deliberately misrepresented in ...
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Status Code 0, or No Code. In my opinion, Arlene Berry's death was premeditated, as evidenced by the physician's documented status "Code 0", and Dr. McLellan knowingly returned a false finding for which he remains to be held to account. From the facts of this case it seems clear that this moral compass isn't such a straight arrow after all.
The Arlene Berry Death Coverup!
In a letter to the College of Physicians and Surgeons of Ontario dated November 28, 2000, Dr. Jordan writes "discussed the situation with family members and a decision was made to intubate Ms. Berry", while the Ambulance Call Report seen at N-7 documents an unsigned patient "Status Code 0", or endorsement of a No Code by proxy. This is an order akin to a 'DNR' denying medical intervention, in this case by passing-the-buck together with all the possible blame.
Patient condition "Status Code Zero" taken from CMAJ articles, is used to describe a "dead" patient condition. The Yale Law Journal, Vol. 93, No. 2 (Dec., 1983), pp. 362-383, criticizes questionable hospital policies "that resort to third party adjudications in No-Code decision making", and provides insight into the assigning of a "no-code order" instructing personnel "not to attempt resuscitation", as in this case. Looking over the chart it is clear that obtaining a 'no code' status in the face of immune mediated adversities by reason of Dr. Jordan's and Dr. Spiller's failure to recognise and treat an emergency situation accordingly, and by reason of Dr. Jordan's failure to attend in a timely manner was the next essential step for these two negligent physicians in executing Arlene Berry's death in order to avoid liability issues.
The Criminal Code of Canada regards euthanasia, whether passive or active, as culpable homicide, or murder: A culpable homicide is defined as murder "where the person who causes the death of a human being means to cause his death" (s. 229, Criminal Code of Canada).
Unless the law has changed in recent years, euthanasia carries a fixed, minimum penalty of 10 years in prison. The 'no code' as evidenced in this case by a "Status Code 0" was ordered by Drs. Jordan, and Spiller, without family knowledge or consent when the patient's condition began to rapidly deteriorate following a whole chain of medical negligence and substandard care that is nothing short of criminal.
With the help of Dr. Mark Arthur Spiller (who admitted Arlene Berry to the Kirkland and District Hospital on the evening of May 23rd), a cross-covering physician working the ER with Dr. Jordan on a rotational basis, these two unscrupulous physicians were able to recruit two of their equally unscrupulous Sudbury colleagues to complete their dirty work at arms length. Interestingly, Dr. Spiller had been a local appointed coroner working under Dr. McLellan at the time, and also a classmate of Dr. Stephane Sauve, one of the Sudbury doctors involved in this medical homicide.
Although it is clear that Arlene Berry was transferred to Sudbury with ventillatory support, and although Drs. Jordan and Spiller were aware of the need for emergency care and life support, after ordering it, they cancelled it, using the secretive no code (Code 0) endorsement as a pretext for evoking a declaration of death and in fact waited for the patient's death.
Within a few hours following her transfer from the Kirkland and District Hospital to the Sudbury Regional Hospital Arlene Berry was declared as having met with 'brain death criteria' , while under the care of Drs. Sauve and Adegbite. Her remains were kept in Sudbury for several days prior to being returned to Kirkland Lake. Withholding life sustaining treatment from an "undiagnosed" patient with concurrent hyperglycemia, hypokalemiaand electrolyte abnormalities in combination with a severely paralysed motor function and who is under the influenceof sedative hypnotic and tranquilizing agents is of questionable legality. Death results from respiratory paralysis and subsequent asphyxiation. Brain death is what happens when ventilator support is discontinued.
Turning off a respirator is a form of passive euthanasia that is practiced by doctors with a family's consent. Turn off the respirator and in the natural course of affairs the patient dies from lack of oxygen. To practice euthenasia by withdrawing life support to a critically ill patient is a medical homicide (to kill or destroy by preventing access of air or oxygen). Passive euthenasia involves an allowing of "nature to take its course". Active euthenasia consists of killing someone (to do acts causing death), or by choosing not to act is also an act, which determines the course and the outcome of events.
An act of wanton and reckless disregard for human life is an act of criminal negligence, in this case, causing death.
In looking over the coroner;s report, was this Dr. McLellan's way of exonerating those responsible for Arlene Berry's death in order to shield the local appointed coroner (working under him at that time) from liability? Or was it just a blatant attempt on his part to shield his fellow colleagues from absolute disgrace?
1. Dr. Sauve sought to open the way to organ donation from brain death under misleading conditions (influence of medications, and metabolic disturbances), further utilizing deception to obtain remote third party consent to harvest my wife's eyes, bypassing permission from the immediate family, namely her defacto spouse and children.
2. Interestingly, Dr. Sauve was a "classmate" of Dr. Mark Spiller, having attended the University of Toronto, Class of '89. Dr. Sauve's name apprears on the Clinical Investigator Inspection List (CLIIL) for Investigational New Drug Studies, and also on the Population Health Research Institute Canada Centre Information list, suggestive of perhaps some hidden agenda, either in the nature of influencing government spending for hospital funding, or some sinister plot involving population control by medical homicide, or both.
3. Suspected of theft of organs for harvesting from a living person by misrepresentation and accessory to medical homicide are: Adegbite, Andrew Babafunso Olanrewaju - Sudbury, ON - (CPSO#54992 ) and Dr. Stephane Jean Sauve - Sudbury (CPSO# 61381); MDs with privileges at the Sudbury Regional Hospital - Laurentian Site.
4. Suspected of criminal negligence causing bodily harm, and liability murder in order to save face are Jordan, Edward Henry - Kirkland Lake, ON (CPSO#61732), and Spiller, Mark Arthur (CPSO#60977); cross-covering physicians working the ER at the Kirkland and District Hospital in Kirkland Lake, ON. In concert with the attending physician, the decision was made by Dr. Jordan to have the patient's senses blunted and prepared for organ harvest.
4. Suspected of conspiracy to conceal an indictable offence(s) and returning a false finding is Dr. Barry Alexander McLellan who so happened to be the Regional Supervising Coroner at the time. Whatsmore, he had been Dr. Spiller's immediate supervisor. A family request for a formal inquest was boldly denied by Dr. McLellan. Interestingly, Dr. McLellan's office was in charge of administering the Human Tissue Gift Act and the Anatomy Act transplants. Dr. McLellan went on to become Ontario's chief coroner and discredited pathologist Charles Smith in order to turn suspicion away from his own faulty findings and sordid immorality. As of September 17, 2007, Dr. McLellan joined Sunnybrook Hospital in Toronto as its new CEO. It is not yet clear whether McLellan stepped down in order to seek refuge among his fellow equally contemptable colleagues at Sunnybrook, or if he was simply given the boot.
This case is now a matter of public record. It has been widely reported and publicized through various internet media, Topix forums as well as the Indymedia IMC throughout Canada, the USA, and the UK. The long standing silence of the doctors named herein confirms their active and ongoing concealment, which includes not only secrecy and deception, but also reticence and non-acknowledgment for their part in the Arlene Berry death cover-up.
The doctors must have intended the death to occur. It is never permissible to take any direct action designed to kill a patient. If a doctor deliberately let a patient die who was suffering from a curable illness, the doctor would certainly be to blame for what he had done, just as he would be to blame if he had needlessly killed the patient. Charges against him would then be appropriate. If so, it would be no defense at all for him to insist that he didn't "do anything." He would have done something very serious indeed, for he let his patient die.
To allow or hasten a patient's death to cover-up error or negligence, or to obtain organs is reprehensible. It is nothing short of murder since such an act or omission which causes death carries the "intended" consequence of the act or omission, hence, the mens rea or criminal intent.
In this case the intent, evidenced by the "Status Code 0" seen at N-7 of the record, is clearly an act of intending to cause the patient's death.
RateMDs.com - Dr. Edward Henry Jordan Doctor Ratings Re: Andrew Adegbite, MD.# 54992 Criminal Negligence Sudbury Regional Hospital - Andrew Adegbite, MD - Topix Dr. A. B. Adegbite - Sudbury, ON Doctor Ratings | RateMDs.com Bacterial Gastrointestinal tract (GIT) infections Infections by Organ System Medical records for attorneys Encephalitis: Diagnosis and Treatment